STAT Stitch Deep Dive Podcast Beyond The Bedside

80/20 High-Yield Emergency Nursing Summary 1. Triage & ESI System Goal : Rapidly categorize patient acuity to treat life threats first[1]. ESI-1 : Immediate lifesaving intervention (e.g., cardiac arrest, intubated trauma, severe anaphylaxis)[1][2]. ESI-2 : High-risk patient , altered mental status, or severe distress (e.g., chest pain, stroke)[2][3]. ESI-3 to 5 : Resource-based (ESI-3 requires...

80/20 High-Yield Emergency Nursing Summary 1. Triage & ESI System Goal : Rapidly categorize patient acuity to treat life threats first[1]. ESI-1 : Immediate lifesaving intervention (e.g., cardiac arrest, intubated trauma, severe anaphylaxis)[1][2]. ESI-2 : High-risk patient , altered mental status, or severe distress (e.g., chest pain, stroke)[2][3]. ESI-3 to 5 : Resource-based (ESI-3 requires...

This episode I go into detail of the current landscape of the US healthcare system and why, you the healthcare practitioner, are under paid and the why your hospital is struggling.

In this episode I discuss the issues with nurse residency programs

This episode details two Pedi Level 1 cases and how important it is for your kids to wear helmets and safety equipment.

According to your sources, Diffuse Axonal Injury (DAI) is widespread axonal damage caused by the physical forces of a mild, moderate, or severe traumatic brain injury (TBI) [1]. Physical Mechanism Traumatic Impact Forces : DAI occurs due to the tensile forces of trauma sustained during a head injury[1]. Cellular Progression : Rather than immediate shearing or tearing of the axon at the moment of i...

Core Intracranial Dynamics (The 20%) Volume Distribution : The enclosed skull holds 78% brain tissue, 12% blood, and 10% CSF [1][2]. Monro-Kellie Doctrine : Intracranial volume is constant; an increase in any component requires compensatory displacement of others to maintain constant volume within the skull[2]. Key Clinical Target Values : Normal ICP is 5–15 mm Hg (sustained >20 mm Hg is abnorm...

An Epidural Hematoma (EDH) and a Subdural Hematoma (SDH) are two distinct forms of focal intracranial bleeding caused by head trauma[1][2]. They differ significantly in anatomical location, primary bleeding source, clinical progression, and at-risk populations[2][3]. 1. Side-by-Side Comparison FeatureEpidural Hematoma (EDH)Subdural Hematoma (SDH)Anatomical Location Between the dura mater and the i...

Core Intracranial Dynamics (The 20%) Volume Distribution : The enclosed skull holds 78% brain tissue, 12% blood, and 10% CSF [1][2]. Monro-Kellie Doctrine : Intracranial volume is constant; an increase in any component requires compensatory displacement of others to maintain constant volume within the skull[2]. Key Clinical Target Values : Normal ICP is 5–15 mm Hg (sustained >20 mm Hg is abnorm...

this episode goes over the 4 kinds of stokes from a high level overview

the intro episode to the clinical judgement model

Burn Classification & TBSA Assessment Burn severity depends on depth, extent (TBSA), location, and patient risk factors[1]. Depth is classified as partial-thickness (epidermal/dermal; painful, red, blistered) or full-thickness (dermal destruction, painless, leathery eschar, visible thrombosed vessels)[2]. TBSA is estimated using the Rule of Nines for adults or the Lund-Browder chart for childr...

Primary Headaches[1] Tension (TTH): Bilateral, dull bandlike pressure[1][2]. No nausea[2]. Tx: NSAIDs[3][4]. Prev: Amitriptyline[3][5]. Migraine: Unilateral throbbing, ± aura[1][6]. Triggers: stress, red wine[6][7]. Acute: Triptans (sumatriptan; contraindicated in CAD, ischemic stroke, uncontrolled HTN)[5]. Prev: Topiramate, β-blockers, Botox[3]. Cluster: Severe unilateral orbital pain in clusters...

80/20 Clinical Stroke Summary Core Concepts & Emergency Recognition A stroke is an emergency where cell death occurs due to ischemia (inadequate blood flow, 87% of cases) or hemorrhage (bleeding, 13%)[1][2]. Interruption of blood flow alters brain metabolism in 30 seconds, stops it in 2 minutes, and causes cell death in 5 minutes[3]. The FAST protocol is critical for rapid recognition: F ace d...

80/20 Clinical Summary: Renal Failure (AKI vs. CKD) 1. Pathophysiology AKI: Sudden, potentially reversible decline in GFR[1]. Prerenal: Decreased perfusion (hypovolemia, shock, HF). Oliguria occurs without tissue damage; reversible with fluids[1][2]. Intrarenal: Direct tissue damage, 90% from Acute Tubular Necrosis (ATN) (ischemia, sepsis, nephrotoxins)[2]. Postrenal: Mechanical obstruction (BPH,...

80/20 Neurological Assessment & Physiology Summary 1. Structure & Cellular Biology Organization: The CNS consists of the brain, spinal cord, and CN I-II[1]. The PNS contains CN III-XII, spinal nerves, and the ANS[1]. Cells & Myelin: Neurons conduct action potentials via saltatory conduction across nodes of Ranvier, accelerated by myelin[2]. Astrocytes form the blood-brain barrier and c...

Clinician’s 80/20 Hematologic & Anemia Summary Core Clinical Concepts Blood disorders disrupt homeostasis. Key pathological threads include: Fatigue : Secondary to tissue hypoxia (anemia, cancer). Infection Risk : Compromised immunity and risk for infection is a major concern in hematologic cancers and a common side effect of therapy. Pain : Common and classic in sickle cell disease (SCD) and...

. DM Classification & Core Diagnostics Type 1 DM : Autoimmune β-cell destruction (HLA-DR3/DR4) causing absolute insulin deficiency; presents abruptly with classic 3 Ps (polyuria, polydipsia, polyphagia), weight loss, and DKA risk[1][2]. Type 2 DM : Insulin resistance combined with relative β-cell deficiency and inappropriate hepatic glucose production[3]. Onset is gradual[4]. Diagnostic Criter...

Urinary System: 80/20 Summary 1. Anatomy & Blood Supply The upper urinary tract has two retroperitoneal, bean-shaped kidneys (T12–L3) and 2 ureters[1]. The lower tract comprises the bladder and urethra[1]. Each kidney is shielded by a fibrous capsule and cushioned by fat[1]. The functional unit is the nephron (~1 million), consisting of a glomerulus, Bowman's capsule, and tubule system (PCT, l...

1. DM Classification & Core Diagnostics Type 1 DM : Autoimmune β-cell destruction (HLA-DR3/DR4) causing absolute insulin deficiency; presents abruptly with classic 3 Ps (polyuria, polydipsia, polyphagia), weight loss, and DKA risk[1][2]. Type 2 DM : Insulin resistance combined with relative β-cell deficiency and inappropriate hepatic glucose production[3]. Onset is gradual[4]. Diagnostic Crite...

1. DM Classification & Core Diagnostics Type 1 DM : Autoimmune β-cell destruction (HLA-DR3/DR4) causing absolute insulin deficiency; presents abruptly with classic 3 Ps (polyuria, polydipsia, polyphagia), weight loss, and DKA risk[1][2]. Type 2 DM : Insulin resistance combined with relative β-cell deficiency and inappropriate hepatic glucose production[3]. Onset is gradual[4]. Diagnostic Crite...

1. DM Classification & Core Diagnostics Type 1 DM : Autoimmune β-cell destruction (HLA-DR3/DR4) causing absolute insulin deficiency; presents abruptly with classic 3 Ps (polyuria, polydipsia, polyphagia), weight loss, and DKA risk[1][2]. Type 2 DM : Insulin resistance combined with relative β-cell deficiency and inappropriate hepatic glucose production[3]. Onset is gradual[4]. Diagnostic Crite...

1. DM Classification & Core Diagnostics Type 1 DM : Autoimmune β-cell destruction (HLA-DR3/DR4) causing absolute insulin deficiency; presents abruptly with classic 3 Ps (polyuria, polydipsia, polyphagia), weight loss, and DKA risk[1][2]. Type 2 DM : Insulin resistance combined with relative β-cell deficiency and inappropriate hepatic glucose production[3]. Onset is gradual[4]. Diagnostic Crite...

80/20 Comparative Summary: Amiodarone vs. Lidocaine 1. Class & Mechanism of Action Amiodarone (Cordarone): Class III antiarrhythmic with Class I, II, III, and IV actions[1][2]. Primarily blocks potassium channels in phases 2 and 3, delaying repolarization and prolonging effective refractory period[1]. Weakly blocks sodium channels (phase 0 upstroke), depresses SA/AV automaticity, blocks calciu...

this is the intro to a new segment comparing two drugs or disease processes to bring you more clarity.