Simini Podcasts
Simini Boards Cast
The Simini Boards-Cast is the go-to audio study tool for small animal surgery residents prepping for board exams. Each episode simplifies high-yield surgical content from trusted sources — built to help you pass faster and with less stress. 🎧 Audio-based learning for passive study ✂️ Practical relevance for surgical application 🧠 Flashcard-style recaps + board-style questions 📈 Designed with resident + program director input Whether you're commuting, walking the dog, or post-op, turn that time into surgical mastery. Subscribe now and get board-ready — fast.
Be sure to visit the podcast's website and support the creator: www.buzzsprout.com
Author
Simini Podcasts
Category
Podcast website
Latest episode
Oct 19, 2026
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Episodes
Chapter 39 - Part A: Force Is Not the Same as Stress: How Bones Experience Load 24.09.2026 20:50
In this BoardsCast episode, we begin Tobias Chapter 39 — Bone Biomechanics and Fracture Biology with a simple but critical distinction: Force happens TO the bone. Stress happens INSIDE the bone. Strain tells you how the bone responds. A 30-kg dog stepping onto a repaired limb does not simply place “30 kg of force” on the femur. Force has magnitude and direction , dynamic ground reaction forces c...
Chapter 39 - Part B: Bone Breaks According to the Load: Reading the Fracture Pattern Backward 24.09.2026 23:30
In this BoardsCast episode, we continue Tobias Chapter 39 — Bone Biomechanics and Fracture Biology with a simple but powerful reframe: A fracture line is the fossilized record of the forces that caused it. A fracture is not a random crack. The pattern on the radiograph reflects the stress environment that existed inside the bone at the moment of failure. We break down the big four loading modes:...
Chapter 39 - Part C: Speed Changes the Failure: Bone Is Stronger Fast, but Breaks Worse 24.09.2026 19:27
In this BoardsCast episode, we continue Tobias Chapter 39 — Bone Biomechanics and Fracture Biology with a biomechanical paradox: Bone can become stronger and stiffer when loaded faster — and still fail worse. The key is strain rate : how fast deformation develops inside the bone. Because bone is viscoelastic, its mechanical behavior changes with loading speed. Slow loading allows more deformatio...
Chapter 39 - Part D: The Gap Is Not the Problem: Interfragmentary Strain Controls What Tissue Can Live 24.09.2026 22:32
In this BoardsCast episode, we continue Tobias Chapter 39 — Bone Biomechanics and Fracture Biology with a counterintuitive idea: The gap is not the problem. The strain environment is the problem. A tiny fracture gap can actually be worse than a larger one if both move the same amount. Why? Because cells do not experience motion alone. They experience motion relative to gap size . That gives us t...
Chapter 39 - Part E: Stability Chooses the Healing Path â Primary vs Secondary Bone Healing 24.09.2026 24:17
In this BoardsCast episode, we finish Tobias Chapter 39 — Bone Biomechanics and Fracture Biology with the idea that ties mechanics and biology together: You do not make bone heal. You create the mechanical and biologic environment that determines how it heals. The same fracture can follow two very different repair programs depending on stability: Absolute stability → primary bone healing Relativ...
Chapter 38 - Part A: The Matrix Is the Machine: Extracellular Matrix and Mechanotransduction 22.09.2026 23:23
In this BoardsCast episode, we jump back to Tobias Chapter 38 — Tissues of the Musculoskeletal System — and rebuild the way you think about connective tissue from the ground up. The central idea: The extracellular matrix is not the packaging. The matrix is the machine. Musculoskeletal tissues don’t behave the way they do simply because of the cells inside them. Their mechanical behavior comes fr...
Chapter 38 - Part B: Bone Is a Living Composite: Why Bone Can Be Hard, Tough, and Adaptive 22.09.2026 22:43
In this BoardsCast episode, we continue Tobias Chapter 38 — Tissues of the Musculoskeletal System by answering a deceptively simple question: How can bone be hard enough to resist compression, tough enough to resist tension, and still remain biologically adaptable? The answer is that bone isn’t rock. Bone is living reinforced concrete. Its mechanical performance comes from a composite partners...
Chapter 38 - Part C: Cartilage Is a Water Mattress Inside a Collagen Net: Compression, Lubrication, and Why Cartilage Fails Differently 22.09.2026 22:25
In this BoardsCast episode, we continue Tobias Chapter 38 — Tissues of the Musculoskeletal System by rebuilding the way you think about articular cartilage. The central mental model: Cartilage is not a rubber pad. It is a water mattress inside a collagen net. Roughly 70% of cartilage is water , while the dry matrix is dominated by type II collagen and proteoglycans like aggrecan . Those proteogl...
Chapter 38 - Part D: Direction Determines Strength: Tendons, Ligaments, and the Tendon-to-Bone Interface 22.09.2026 18:18
In this BoardsCast episode, we continue Tobias Chapter 38 — Tissues of the Musculoskeletal System with the rule that explains why tendons and ligaments can be incredibly strong in one direction and surprisingly vulnerable in another: Direction determines strength. Tendons and ligaments are anisotropic tissues —their mechanical properties depend on the direction in which they’re loaded. Their typ...
Chapter 38 - Part E: Muscle Makes Force. Connective Tissue Delivers It: Contraction, Force Transmission, and Regeneration 22.09.2026 20:19
In this BoardsCast episode, we finishTobias Chapter 38 — Tissues of the Musculoskeletal System with a simple but powerful reframe: The sarcomere is the engine. Connective tissue is the transmission. A muscle can generate enormous internal force and still fail to create movement if that force never reaches the tendon and skeleton. That’s the core idea of this episode: Muscle makes force. Connecti...
Chapter 125 - Part A: The Eye Is a Machine Under Pressure 11.06.2026 20:49
In this BoardsCast episode, we begin Tobias Chapter 125 — Emergency Ophthalmic Surgery with the single mental model that decides outcomes in ocular trauma: The eye is a sealed pressure vessel. You can have a clear cornea, an attached retina, and an intact optic nerve… and still have a blind eye—because the container failed. This episode reframes acute vision loss as a mechanical problem first: if...
Chapter 125 - Part B: Save the Wall Before You Save the Vision 11.06.2026 20:46
In this BoardsCast episode, we continue Tobias Chapter 125 — Emergency Ophthalmic Surgery with the single mindset shift that saves globes: Stop thinking like a photographer fixing a lens. Start thinking like a structural engineer stabilizing a collapsing wall. A melting corneal ulcer isn’t a “vision problem.” It’s a structural failure in progress . The stroma (the load-bearing collagen wall) is be...
Chapter 125 - Part C: The Outside Protects the Inside 11.06.2026 17:01
In this BoardsCast episode, we continue Tobias Chapter 125 — Emergency Ophthalmic Surgery by ignoring the inside of the eye on purpose. Because none of it matters if the defenses fail. This episode installs one core mental model: The ocular surface survives only when its protective structures remain functional; the outside protects the inside. We break down the “castle wall” system—eyelids, conj...
Chapter 125 - Part D: The Eye Is an Optical Alignment System 11.06.2026 20:41
In this BoardsCast episode, we continue Tobias Chapter 125 — Emergency Ophthalmic Surgery with the mental model that turns “random eye emergencies” into predictable mechanics: The eye is not just living tissue. It’s an optical alignment system. You can have a clear lens, a viable retina, and an intact cornea… and still have a nonfunctional eye—because the parts are in the wrong place. This episode...
Chapter 125 - Part E: Tissue Dies on a Clock 11.06.2026 20:51
In this BoardsCast episode, we finish Tobias with Chapter 125 — Emergency Ophthalmic Surgery — the final chapter of Tobias and the final episode in this textbook series. And we end with the one mental model that upgrades how you triage every eye emergency: The eye is not waiting. The clock is already running. Some ophthalmic problems aren’t “diagnose then schedule.” They’re active tissue-destructi...
Chapter 124 - Part A: The Eye Is a Microsurgery Environment 03.06.2026 15:52
In this BoardsCast episode, we begin Tobias Chapter 124 — Basic Ophthalmic Surgical Procedures with the only mental model that actually predicts outcomes in eye surgery: Ophthalmic surgery succeeds or fails before the first incision. Because the eye is not a “standard surgical field.” It’s a micro-environment where tiny mistakes become immediate disasters: corneal drying, harsh prep solutions, poo...
Chapter 124 - Part B: The Eyelid Is Not Skin 03.06.2026 22:19
In this BoardsCast episode, we continue Tobias Chapter 124 — Basic Ophthalmic Surgical Procedures with the surgical paradox that ruins eyes: The incision can look perfect… and the cornea can still ulcerate. Because the eyelid is not skin. It’s a moving protective machine —a windshield wiper for the cornea. And in eyelid surgery, cosmetic closure is irrelevant if the “wiper blade” doesn’t glide per...
Chapter 124 - Part C: The Ocular Surface Maintenance System 03.06.2026 19:37
In this BoardsCast episode, we continue Tobias Chapter 124 — Basic Ophthalmic Surgical Procedures with the framework that explains why an eye can look “structurally normal” and still ulcerate: The eye doesn’t stay healthy because it has tears. It stays healthy because tears move. This episode breaks ocular surface health into the three pillars that must run continuously: Production: the tear film...
Chapter 124 - Part D: Save the Surface: Why Corneal Surgery Exists 03.06.2026 20:54
In this BoardsCast episode, we continue Tobias Chapter 124 — Basic Ophthalmic Surgical Procedures with the one corneal truth that changes everything: You can treat infection. You can control inflammation. You can manage pain. But if the cornea disappears, none of it matters—because the front wall of the eye is gone. This is why corneal surgery exists. Not for “vision correction.” Not for cosmetic...
Chapter 124 - Part E: When the Eye Cannot Be Saved 03.06.2026 20:09
In this BoardsCast episode, we finish Tobias Chapter 124 — Basic Ophthalmic Surgical Procedures with the hardest (and highest-level) decision in ophthalmic surgery: When do you stop saving the globe… and start saving the patient from the globe? This episode builds a clear, board-ready decision framework for end-stage eyes that are blind, painful, and failing —where continued “salvage” is no longer...
Chapter 123 - Part A: Why We Can Hear: The Sound Transmission Machine 02.06.2026 21:26
In this BoardsCast episode, we begin Tobias Chapter 123 — Middle and Inner Ear with the core reframe that makes hearing loss finally make sense: Hearing isn’t a sensing problem first — it’s a transfer problem. The cochlea can be normal. The nerve can be intact. The brain can be fine. And the patient can still be deaf — because sound energy never makes it across the air-to-fluid border. This episo...
Chapter 123 - Part B: Why Ears Stay Healthy: Drainage, Pressure, and Clearance 02.06.2026 20:57
In this BoardsCast episode, we continue Tobias Chapter 123 — Middle and Inner Ear by flipping the usual middle ear assumption on its head: A healthy middle ear isn’t healthy because it’s sterile. It’s healthy because it drains. The tympanic cavity constantly produces secretions and debris. If it can’t clear them, it fills—sometimes with mucus even when there’s no tumour, no rupture, and no infecti...
Chapter 123 - Part C: Why Otitis Media Happens: The Three Routes of Invasion 02.06.2026 19:22
In this BoardsCast episode, we continue Tobias Chapter 123 — Middle and Inner Ear by killing the most common wrong question in otitis media: You cultured bacteria. You found pus in the bulla. Everyone asks what bug it is. Wrong question. The real question is: how did it get there? Because pathogens don’t teleport. Middle ear infection is a logistics problem : a sealed bony vault was breached, and...
Chapter 123 - Part D: When Ear Disease Becomes Neurologic Disease 02.06.2026 13:22
In this BoardsCast episode, we continue Tobias Chapter 123 — Middle and Inner Ear with the highest-stakes clinical transition in otitis: The disease didn’t change. The ZIP code did. A patient walks in with “an ear infection”… and suddenly has a head tilt, facial droop, Horner syndrome, and ataxia. That’s not because the bacteria got smarter — it’s because middle ear disease lives next to critical...
Chapter 123 - Part E: Why Ear Surgery Works: Remove the Disease Reservoir 02.06.2026 17:34
In this BoardsCast episode, we finish Tobias Chapter 123 — Middle and Inner Ear with the core reason chronic ear cases relapse: You killed the tenants… but you left the apartment complex standing. This is the mental model that drives the entire episode: Medicine controls infection. Surgery removes the environment that creates it. We break down why systemic and topical antimicrobials can suppress o...
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